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Contributor Information

  • Name Nigel Dimmock
  • Institute University of Warwick

Tool Details

  • Tool name: Anti-HA (H1N1) [LM41]
  • Alternate names: Hemagglutinin Antibody, Anti-HA, HA, Antibody, Anti-Influenza A, Anti-H1N1 HA, Influenza A Antibody, Flu Antibody, AWSN Antibody, H1N1 Antibody, Anti-AWSN, A/WSN Antibody, Anti-A/WSN
  • Clone: LM41
  • Tool type: Antibodies
  • Class: Monoclonal
  • Conjugate: Unconjugated
  • Reactivity: Virus
  • Host: Mouse
  • Application: ELISA ; Fn
  • Description: The hemagglutinin (HA) protein is one of two major surface glycoproteins on the envelope of influenza A virus. The HA protein is responsible for receptor binding to host cells and for viral entry and is therefore the primary target of neutralizing antibodies. LM41 recognises an epitope of the A/WSN (H1N1) HA antigen only when expressed in H-2k cells. LM41 inhibited Ag-specific, MHC class I-restricted lysis of H-2k target cells infected with a vaccinia recombinant virus expressing the HA of A/WSN by CTL from A/WSN virus-infected H-2k mice.
  • Immunogen: C3WHe-mg (H-2k) mice were infected with A/WSN (H1N1)
  • Isotype: IgM
  • Research area: Immunology; Microbiology
  • Myeloma used: Sp2/0-Ag14

  • For Research Use Only

Target Details

  • Target: A/WSN (H1N1) hemagglutinin
  • Target background: The hemagglutinin (HA) protein is one of two major surface glycoproteins on the envelope of influenza A virus. The HA protein is responsible for receptor binding to host cells and for viral entry and is therefore the primary target of neutralizing antibodies. LM41 recognises an epitope of the A/WSN (H1N1) HA antigen only when expressed in H-2k cells. LM41 inhibited Ag-specific, MHC class I-restricted lysis of H-2k target cells infected with a vaccinia recombinant virus expressing the HA of A/WSN by CTL from A/WSN virus-infected H-2k mice.

Application Details

  • Application: ELISA ; Fn

Handling

  • Format: Liquid
  • Storage buffer:
  • Storage conditions:
  • Shipping conditions: Shipping at 4°C

Documentation

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References

  • •  McLain et al. 1993. J Immunol. 150(8 Pt 1):3421-6. PMID: 7682240.